"This trick would not eliminate the virus, but it might be able to permanently raise the T-cell counts of AIDS patients, increasing their ability to resist secondary infections and remain healthy." link they're already using entry inhibitors to suppress/block binding to ccr5. this mechanism is different. it would be a mutation of ccr5.
although this is the major receptor, hiv uses other receptors for entry too. as seen with so many hiv drugs and therapies, you're selecting for those viruses that can escape the ccr5 mutation. and you may block other necessary traffic that uses ccr5. you'd still need a combination therapy. it'll be some years before it makes it to people. curious to see the outcome.
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